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* {{cite journal |author=Hardy MP |title=Characterization of the type I interferon locus and identification of novel genes |journal=Genomics |volume=84 |issue= 2 |pages= 331–45 |year= 2004 |pmid= 15233997 |doi= 10.1016/j.ygeno.2004.03.003 |author-separator=, |author2=Owczarek CM |author3=Jermiin LS |display-authors=3 |last4=Ejdebäck |first4=Mikael |last5=Hertzog |first5=Paul J. }}
* {{cite journal |author=Hardy MP |title=Characterization of the type I interferon locus and identification of novel genes |journal=Genomics |volume=84 |issue= 2 |pages= 331–45 |year= 2004 |pmid= 15233997 |doi= 10.1016/j.ygeno.2004.03.003 |author-separator=, |author2=Owczarek CM |author3=Jermiin LS |display-authors=3 |last4=Ejdebäck |first4=Mikael |last5=Hertzog |first5=Paul J. }}
* {{cite journal |author=Silva LK |title=Dengue hemorrhagic fever is associated with polymorphisms in JAK1 |journal=Eur. J. Hum. Genet. |volume=18 |issue= 11 |pages= 1221–7 |year= 2010 |pmid= 20588308 |doi= 10.1038/ejhg.2010.98 |pmc=2950898 |author-separator=, |author2=Blanton RE |author3=Parrado AR |display-authors=3 |last4=Melo |first4=Paulo S |last5=Morato |first5=Vanessa G |last6=Reis |first6=Eliana AG |last7=Dias |first7=Juarez P |last8=Castro |first8=Jesuina M |last9=Vasconcelos |first9=Pedro FC}}
* {{cite journal |author=Silva LK |title=Dengue hemorrhagic fever is associated with polymorphisms in JAK1 |journal=Eur. J. Hum. Genet. |volume=18 |issue= 11 |pages= 1221–7 |year= 2010 |pmid= 20588308 |doi= 10.1038/ejhg.2010.98 |pmc=2950898 |author-separator=, |author2=Blanton RE |author3=Parrado AR |display-authors=3 |last4=Melo |first4=Paulo S |last5=Morato |first5=Vanessa G |last6=Reis |first6=Eliana AG |last7=Dias |first7=Juarez P |last8=Castro |first8=Jesuina M |last9=Vasconcelos |first9=Pedro FC}}
* {{cite journal |author=Janssen R |title=Genetic susceptibility to respiratory syncytial virus bronchiolitis is predominantly associated with innate immune genes |journal=J. Infect. Dis. |volume=196 |issue= 6 |pages= 826–34 |year= 2007 |pmid= 17703412 |doi= 10.1086/520886 |author-separator=, |author2=Bont L |author3=Siezen CL |display-authors=3 |last4=Hodemaekers |first4=Hennie M. |last5=Ermers |first5=Marieke J. |last6=Doornbos |first6=Gerda |last7=Slot |first7=Ruben van ’t |last8=Wijmenga |first8=Ciska |last9=Goeman |first9=Jelle J. }}
* {{cite journal |author=Janssen R |title=Genetic susceptibility to respiratory syncytial virus bronchiolitis is predominantly associated with innate immune genes |journal=J. Infect. Dis. |volume=196 |issue= 6 |pages= 826–34 |year= 2007 |pmid= 17703412 |doi= 10.1086/520886 |author-separator=, |author2=Bont L |author3=Siezen CL |display-authors=3 |last4=Hodemaekers |first4=Hennie M. |last5=Ermers |first5=Marieke J. |last6=Doornbos |first6=Gerda |last7=Slot |first7=Ruben van ’t |last8=Wijmenga |first8=Ciska |last9=Goeman |first9=Jelle J. }}
* {{cite journal |author=Shuai K |title=Interferon activation of the transcription factor Stat91 involves dimerization through SH2-phosphotyrosyl peptide interactions |journal=Cell |volume=76 |issue= 5 |pages= 821–8 |year= 1994 |pmid= 7510216 |doi= 10.1016/0092-8674(94)90357-3 |author-separator=, |author2=Horvath CM |author3=Huang LH |display-authors=3 |last4=Qureshi |first4=SA |last5=Cowburn |first5=D |last6=Darnell Jr |first6=JE }}
* {{cite journal |author=Shuai K |title=Interferon activation of the transcription factor Stat91 involves dimerization through SH2-phosphotyrosyl peptide interactions |journal=Cell |volume=76 |issue= 5 |pages= 821–8 |year= 1994 |pmid= 7510216 |doi= 10.1016/0092-8674(94)90357-3 |author-separator=, |author2=Horvath CM |author3=Huang LH |display-authors=3 |last4=Qureshi |first4=SA |last5=Cowburn |first5=D |last6=Darnell Jr |first6=JE }}
* {{cite journal |author=Gerhard DS |title=The status, quality, and expansion of the NIH full-length cDNA project: the Mammalian Gene Collection (MGC) |journal=Genome Res. |volume=14 |issue= 10B |pages= 2121–7 |year= 2004 |pmid= 15489334 |doi= 10.1101/gr.2596504 |pmc=528928 |author-separator=, |author2=Wagner L |author3=Feingold EA |display-authors=3 |last4=Shenmen |first4=CM |last5=Grouse |first5=LH |last6=Schuler |first6=G |last7=Klein |first7=SL |last8=Old |first8=S |last9=Rasooly |first9=R}}
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Revisi terkini sejak 24 November 2018 12.53

Interferon, alpha 13
Struktur tersedia
PDB Ortholog search: PDBe, RCSB
Tanda pengenal
Simbol IFNA13; IFNA1
ID eksternal OMIM147578 MGI3641425 HomoloGene68536 GeneCards: IFNA13 Gene
Orthologs
Spesies Manusia Tikus
Entrez 3447 15962
Ensembl ENSG00000233816 ENSMUSG00000095498
UniProt P01562 P01572
RefSeq (mRNA) NM_006900 NM_010502
RefSeq (protein) NP_008831 NP_034632
Lokasi (UCSC) Chr 9:
21.37 – 21.37 Mb
Chr 4:
88.85 – 88.85 Mb
PubMed [1] [2]

Interferon alpha-1/13, juga dikenal sebagai IFN-alpha-1/13, adalah protein yang pada tubuh manusia disandikan oleh gen IFNA1 dan IFNA13. IFNA13 adalah gen interferon.[1][2]

Referensi[sunting | sunting sumber]

  1. ^ "Entrez Gene: interferon". 
  2. ^ Olopade OI, Bohlander SK, Pomykala H, Maltepe E, Van Melle E, Le Beau MM, Diaz MO (1992). "Mapping of the shortest region of overlap of deletions of the short arm of chromosome 9 associated with human neoplasia". Genomics. 14 (2): 437–43. doi:10.1016/S0888-7543(05)80238-1. PMID 1385305. 

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